Data Entry: Please note that the research database will be replaced by UNIverse by the end of October 2023. Please enter your data into the system https://universe-intern.unibas.ch. Thanks

Login for users with Unibas email account...

Login for registered users without Unibas email account...

 
Effects of plantarflexion on pelvis and lower limb kinematics
JournalArticle (Originalarbeit in einer wissenschaftlichen Zeitschrift)
 
ID 1194758
Author(s) Brunner, R; Dreher, T; Romkes, J; Frigo, C
Author(s) at UniBasel Brunner, Reinald G.H.
Year 2008
Title Effects of plantarflexion on pelvis and lower limb kinematics
Journal Gait Posture
Volume 28
Number 1
Pages / Article-Number 150-6
Keywords soleus gastrocnemius function, biomechanical modelling, hip internal rotation, pelvic retraction, spastic plantarflexion
Abstract Modelling the effect of soleus and gastrocnemius contractions against the floor resistance in a forward dynamics simulation revealed that hip flexion, internal rotation and adduction together with external pelvic rotation could be attributed to a direct, but distant effect of triceps surae contraction. Knee flexion smoothed out the effect. To validate this clinically relevant biomechanical observation, ankle plantar flexion was correlated with hip and pelvic rotation retrospectively in children with spastic cerebral palsy. In 49 children with spastic hemiplegia, plantar flexion showed a significant correlation with increased pelvic retraction and hip internal rotation. In contrast, in 47 children with spastic diplegia no significant effect of the triceps surae on hip and pelvis kinematics was found. Bilateral hip and knee flexion in diplegia appeared to prevent the proximal effect of the triceps surae seen in the hemiplegics. In diplegia triceps surae overactivity did not appear to be a significant cause of internal rotation gait.
Publisher Elsevier
ISSN/ISBN 0966-6362
edoc-URL http://edoc.unibas.ch/dok/A6004961
Full Text on edoc No
Digital Object Identifier DOI 10.1016/j.gaitpost.2007.11.013
PubMed ID http://www.ncbi.nlm.nih.gov/pubmed/18258430
ISI-Number WOS:000257019600022
Document type (ISI) Journal Article
 
   

MCSS v5.8 PRO. 0.366 sec, queries - 0.000 sec ©Universität Basel  |  Impressum   |    
06/05/2024